ROBINUL glycopyrronium bromide (glycopyrrolate) 0.2mg/1mL injection ampoule Australia - English - Department of Health (Therapeutic Goods Administration)

robinul glycopyrronium bromide (glycopyrrolate) 0.2mg/1ml injection ampoule

aspen pharmacare australia pty ltd - glycopyrronium bromide, quantity: 0.2 mg/ml - injection, solution - excipient ingredients: water for injections; sodium hydroxide; sodium chloride; hydrochloric acid - 1.as a preoperative antimuscarinic to reduce salivary, tracheobronchial and pharyngeal secretions, and to reduce the acidity and volume of the gastric contents. 2.as a preoperative or intraoperative antimuscarinic to attenuate or prevent intraoperative bradycardia associated with the use of suxa-methonium or due to cardiac vagal reflexes. 3.to protect against the peripheral muscarinic actions (eg. bradycardia and excessive secretions) of anticholinesterases such as neostigmine or pyridostigmine given to reverse neuromuscular blockade produced by nondepolarising muscle relaxants.

Robinul New Zealand - English - Medsafe (Medicines Safety Authority)

robinul

pharmacy retailing (nz) ltd t/a healthcare logistics - glycopyrronium bromide 0.2 mg/ml; glycopyrronium bromide 0.2 mg/ml - solution for injection - 0.2 mg/ml - active: glycopyrronium bromide 0.2 mg/ml excipient: hydrochloric acid sodium chloride sodium hydroxide water for injection active: glycopyrronium bromide 0.2 mg/ml excipient: benzyl alcohol hydrochloric acid water for injection - robinul is indicated for use as a preoperative antimuscarinic to reduce salivary, tracheobronchial and pharyngeal secretions; to reduce the volume and free acidity of gastric secretions, and to block cardiac vagal inhibitory reflexes during induction of anaesthesia and intubation when indicated. robinul injectable may be used intraoperatively to counteract drug-induced or vagal traction reflexes with the associated arrhythmias. glycopyrrolate protects against the peripheral muscarinic effects (e.g. bradycardia and excessive secretions) of cholinergic agents such as neostigmine and pyridostigmine given to reverse the neuromuscular blockade due to non-depolarising muscle relaxants.

GLYCOPYRROLATE ACCORD glycopyrrolate 0.2 mg/1 mL injection ampoules Australia - English - Department of Health (Therapeutic Goods Administration)

glycopyrrolate accord glycopyrrolate 0.2 mg/1 ml injection ampoules

accord healthcare pty ltd - glycopyrronium bromide, quantity: 0.2 mg/ml - injection, solution - excipient ingredients: water for injections; hydrochloric acid; sodium chloride - 1. as a preoperative antimuscarinic to reduce salivary, tracheobronchial and pharyngeal secretions, and to reduce the acidity and volume of the gastric contents.,2. as a preoperative or intra-operative antimuscarinic to attenuate or prevent intra- operative bradycardia associated with the use of suxamethonium or due to cardiac vagal reflexes.,3. to protect against the peripheral muscarinic actions (e.g. bradycardia and excessive secretions) of anticholinesterases such as neostigmine or pyridostigmine given to reverse neuromuscular blockade produced by non-depolarising muscle relaxants.

GLYCOPYRROLATE ACCORD glycopyrrolate 0.6 mg/3 mL injection ampoules Australia - English - Department of Health (Therapeutic Goods Administration)

glycopyrrolate accord glycopyrrolate 0.6 mg/3 ml injection ampoules

accord healthcare pty ltd - glycopyrronium bromide, quantity: 0.2 mg/ml - injection, solution - excipient ingredients: water for injections; sodium chloride; hydrochloric acid - 1. as a preoperative antimuscarinic to reduce salivary, tracheobronchial and pharyngeal secretions, and to reduce the acidity and volume of the gastric contents.,2. as a preoperative or intra-operative antimuscarinic to attenuate or prevent intra- operative bradycardia associated with the use of suxamethonium or due to cardiac vagal reflexes.,3. to protect against the peripheral muscarinic actions (e.g. bradycardia and excessive secretions) of anticholinesterases such as neostigmine or pyridostigmine given to reverse neuromuscular blockade produced by non-depolarising muscle relaxants.